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Merck

W322318

Phenol

≥99%

Synonym(s):

Hydroxybenzene

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About This Item

Linear Formula:
C6H5OH
CAS Number:
Molecular Weight:
94.11
FEMA Number:
3223
Council of Europe no.:
11811
UNSPSC Code:
12164502
PubChem Substance ID:
Flavis number:
4.041
EC Number:
203-632-7
NACRES:
NA.21
MDL number:
Beilstein/REAXYS Number:
969616
Organoleptic:
phenolic
Grade:
Halal
Kosher
Biological source:
synthetic
Agency:
meets purity specifications of JECFA
Food allergen:
no known allergens
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Product Name

Phenol, ≥99%

SMILES string

Oc1ccccc1

InChI

1S/C6H6O/c7-6-4-2-1-3-5-6/h1-5,7H

InChI key

ISWSIDIOOBJBQZ-UHFFFAOYSA-N

biological source

synthetic

grade

Halal
Kosher

agency

meets purity specifications of JECFA

vapor density

3.24 (vs air)

vapor pressure

0.09 psi ( 55 °C)
0.36 mmHg ( 20 °C)

assay

≥99%

autoignition temp.

1319 °F

may contain

≤0.15% hypophosphorous acid as additive

expl. lim.

8.6 %

bp

182 °C (lit.)

mp

40-42 °C (lit.)

density

1.071 g/mL at 25 °C (lit.)

application(s)

flavors and fragrances

documentation

see Safety & Documentation for available documents

food allergen

no known allergens

organoleptic

phenolic

Quality Level

Gene Information

human ... GABRA1(2554)

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Related Categories

Application


  • Revealing mechanism of phenolic-amine reaction to form humus in compost based on high-resolution liquid chromatography mass spectrometry and spectroscopy.: This study delves into the complex reactions between phenolic compounds and amines during compost formation, shedding light on the molecular pathways that lead to humus production, crucial for soil fertility and carbon sequestration (Zhou et al., 2024).

  • Unlocking the potential of phenolated kraft lignin as a versatile feed additive.: This paper explores the utilization of phenolated kraft lignin as a feed additive, examining its benefits in animal nutrition and its role in sustainable agricultural practices (Li et al., 2024).

Disclaimer

For R&D or non-EU Food use. Not for retail sale.

signalword

Danger

Hazard Classifications

Acute Tox. 3 Dermal - Acute Tox. 3 Inhalation - Acute Tox. 3 Oral - Aquatic Chronic 2 - Eye Dam. 1 - Muta. 2 - Skin Corr. 1B - STOT RE 2

target_organs

Nervous system,Kidney,Liver,Skin

Storage Class

6.1A - Combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials

wgk

WGK 2

flash_point_f

177.8 °F - closed cup

flash_point_c

81 °C - closed cup

ppe

Eyeshields, Faceshields, Gloves, type P3 (EN 143) respirator cartridges


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Thitiporn Anunthawan et al.
Biochimica et biophysica acta, 1848(6), 1352-1358 (2015-03-15)
We investigated the mechanisms of two tryptophan-rich antibacterial peptides (KT2 and RT2) obtained in a previous optimization screen for increased killing of both Gram-negative and Gram-positive bacteria pathogens. At their minimal inhibitory concentrations (MICs), these peptides completely killed cells of
Rita A Busuttil et al.
Clinical cancer research : an official journal of the American Association for Cancer Research, 20(10), 2761-2772 (2014-03-25)
Gene-expression profiling has revolutionized the way we think about cancer and confers the ability to observe the synchronous expression of thousands of genes. The use of putative genome-level expression profiles has allowed biologists to observe the complex interactions of genes
Mark J Lee et al.
PLoS pathogens, 11(10), e1005187-e1005187 (2015-10-23)
Of the over 250 Aspergillus species, Aspergillus fumigatus accounts for up to 80% of invasive human infections. A. fumigatus produces galactosaminogalactan (GAG), an exopolysaccharide composed of galactose and N-acetyl-galactosamine (GalNAc) that mediates adherence and is required for full virulence. Less
Saori Tomita et al.
Nature communications, 6, 6966-6966 (2015-04-30)
Estrogen receptor-α (ER)-positive breast cancer cells undergo hormone-independent proliferation after deprivation of oestrogen, leading to endocrine therapy resistance. Up-regulation of the ER gene (ESR1) is critical for this process, but the underlying mechanisms remain unclear. Here we show that the
Jong-Won Lim et al.
Human molecular genetics, 24(17), 4817-4828 (2015-06-05)
Facioscapulohumeral muscular dystrophy (FSHD) is caused by the aberrant expression of the DUX4 transcription factor in skeletal muscle. The DUX4 retrogene is encoded in the D4Z4 macrosatellite repeat array, and smaller array size or a mutation in the SMCHD1 gene

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