85L2
Leukocyte Alkaline Phosphatase Kit
based on naphthol AS-MX phosphate and fast blue RR salt (with citrate)
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About This Item
NACRES:
NA.47
UNSPSC Code:
12352106
shelf life
Expiry date on the label.
IVD
for in vitro diagnostic use
dilution
(for histology)
application(s)
hematology
histology
shipped in
wet ice
storage temp.
2-8°C
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Application
Leukocyte Alkaline Phosphatase (LAP) kits are intended for the qualitative demonstration of alkaline phosphatase activity in white blood cells.
Peripheral blood or bone marrow preparations are fixed to a microscope slide. The film is then incubated in a mixture of naphthol AS-MX phosphate alkaline solution with fast blue RR salt. The resulting blue, insoluble, granular dye deposit indicates sites of alkaline phosphatase activity.
signalword
Warning
hcodes
Hazard Classifications
Eye Irrit. 2 - Met. Corr. 1 - STOT SE 3
target_organs
Respiratory system
Storage Class
10 - Combustible liquids
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Guenaelle Bouet et al.
PloS one, 10(2), e0117402-e0117402 (2015-02-25)
Bone sialoprotein (BSP) belongs to the "small integrin-binding ligand N-linked glycoprotein" (SIBLING) family, whose members interact with bone cells and bone mineral. BSP is strongly expressed in bone and we previously showed that BSP knockout (BSP-/-) mice have a higher
G Bouet et al.
European cells & materials, 29, 250-266 (2015-05-16)
An engineered three dimensional (3D) in vitro cell culture system was designed with the goal of inducing and controlling in vitro osteogenesis in a reproducible manner under conditions more similar to the in vivo bone microenvironment than traditional two-dimensional (2D)
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Developmental cell, 14(6), 914-925 (2008-06-10)
Maintenance of bone mass and integrity requires a tight balance between resorption by osteoclasts and formation by osteoblasts. Exocytosis of functional proteins is a prerequisite for the activity of both cells. In the present study, we show that synaptotagmin VII
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Transcriptional regulation plays an essential role in the self-renewal and differentiation of human embryonic stem cells (hESCs). However, how external signals disrupt the self-renewal regulatory network and further drive hESC differentiation remains largely unknown. Here, we found the immune regulative
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