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Merck

911127

Ammonium persulfate

free-flowing, Redi-Dri™, reagent grade, 98%

Synonym(s):

AP, APS, Ammonium peroxodisulfate, Ammonium peroxydisulfate, PER

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About This Item

Linear Formula:
(NH4)2S2O8
CAS Number:
7727-54-0
Molecular Weight:
228.20
UNSPSC Code:
12352300
NACRES:
NA.21
MDL number:
MFCD00003390
Assay:
98%
Grade:
reagent grade
Form:
powder

Key Documents

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InChI

1S/2H3N.H2O8S2/c;;1-9(2,3)7-8-10(4,5)6/h2*1H3;(H,1,2,3)(H,4,5,6)

SMILES string

N.N.OS(=O)(=O)OOS(O)(=O)=O

InChI key

ROOXNKNUYICQNP-UHFFFAOYSA-N

grade

reagent grade

vapor density

7.9 (vs air)

product line

Redi-Dri™

assay

98%

form

powder

pH

1-2 (25 °C, 228 g/L)

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Application

Redi-Dri™ is an innovative packaging product line that helps eliminate clumping material by adding extra packaging process steps without any chemical additives, anti-clumping agents, or hydrophobic compounds. Redi-Dri™ offers a more convenient and safer way to handle the products.
Catalyst for acrylamide gel polymerization.

Legal Information

Redi-Dri is a trademark of Sigma-Aldrich Co. LLC

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Danger

Hazard Classifications

Acute Tox. 4 Oral - Eye Irrit. 2 - Ox. Sol. 3 - Resp. Sens. 1 - Skin Irrit. 2 - Skin Sens. 1 - STOT SE 3

target_organs

Respiratory system

Storage Class

5.1B - Oxidizing hazardous materials

wgk

WGK 1

flash_point_f

Not applicable

flash_point_c

Not applicable

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Certificates of Analysis (COA)

Lot/Batch Number
MKCV9705
MKCV1966
MKCT5190
MKCK3540

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Karin Taylor et al.
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Journal of proteomics, 130, 26-32 (2015-09-16)
Definition of the urinary protein composition would represent a potential tool for diagnosis in many clinical conditions. The use of new proteomic technologies allows detection of genetic and post-trasductional variants that increase sensitivity of the approach but complicates comparison within
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Molecular biology of the cell, 25(25), 4115-4129 (2014-10-31)
The vascular endothelium is a highly dynamic structure, and the integrity of its barrier function is tightly regulated. Normally impenetrable to cells, the endothelium actively assists lymphocytes to exit the bloodstream during inflammation. The actin cytoskeleton of the endothelial cell
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Nature materials, 14(3), 343-351 (2015-02-11)
The origin of fracture in epithelial cell sheets subject to stretch is commonly attributed to excess tension in the cells' cytoskeleton, in the plasma membrane, or in cell-cell contacts. Here, we demonstrate that for a variety of synthetic and physiological
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