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Merck

MAB5628

Anti-Glial Fibrillary Acidic Protein Antibody

CHEMICON®, mouse monoclonal,

Synonyme(s) :

GFAP

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A propos de cet article

UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

Principaux documents

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Nom du produit

Anti-Glial Fibrillary Acidic Protein Antibody, Chemicon®, from mouse

biological source

mouse

antibody form

purified immunoglobulin

antibody product type

primary antibodies

clone

monoclonal

species reactivity

rat, human

manufacturer/tradename

Chemicon®

technique(s)

immunocytochemistry: suitable
western blot: suitable

isotype

IgG2a

NCBI accession no.

NM_002055.2

UniProt accession no.

P14136

shipped in

wet ice

target post-translational modification

unmodified

Quality Level

100

Gene Information

human ... GFAP(2670)

Analysis Note

Control
Positive: Neural tissue or astrocyte culture
Negative: Non-neural tissue or neuronal culture

Application

Detect Glial Fibrillary Acidic Protein using this Anti-Glial Fibrillary Acidic Protein Antibody validated for use in WB & IC.
Western blot: 1:10,000 on rat brain

Optimal working dilutions must be determined by end user.

Other Notes

Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.
Replaces: 04-1031; 04-1062

Physical form

Format: Purified
Purified immunoglobulin. Liquid in 0.02M phosphate buffer, 0.25 M NaCl, pH 7.6 with 0.1% sodium azide.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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Classe de stockage

10 - Combustible liquids

wgk

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable

Certificats d'analyse (COA)

Recherchez un Certificats d'analyse (COA) en saisissant le numéro de lot du produit. Les numéros de lot figurent sur l'étiquette du produit après les mots "Lot" ou "Batch".

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Takashi Ishijima et al.
Science progress, 104(4), 368504211054985-368504211054985 (2021-11-26)
By using an animal model in which inflammatory cytokines are induced in lipopolysaccharide (LPS)-injected rat brain, we investigated the induction of tumor necrosis factor alpha (TNFα), interleukin-1beta (IL-1β), and IL-6. Immunoblotting and immunohistochemistry revealed that all three cytokines were transiently
Sitthisak Thongrong et al.
Biomedical reports, 18(3), 20-20 (2023-02-18)
Chronic stress has been recognized to induce the alterations of neuronal and glial cells in the hippocampus, and is thus implicated in cognitive dysfunction. There is increasing evidence to indicate that natural compounds capable of exerting neuroprotective and antioxidant activities
Shun-Yun Cheng et al.
Molecular therapy. Nucleic acids, 35(1), 102088-102088 (2024-01-09)
Inherited retinal dystrophies caused by dominant mutations in photoreceptor (PR) cell expressed genes are a major cause of irreversible vision loss. Oligonucleotide therapy has been of interest in diseases that conventional medicine cannot target. In the early days, small interfering
Heather R Minkel et al.
Glia, 63(12), 2285-2297 (2015-07-21)
Alexander Disease (AxD) is a "gliopathy" caused by toxic, dominant gain-of-function mutations in the glial fibrillary acidic protein (GFAP) gene. Two distinct types of AxD exist. Type I AxD affected individuals develop cerebral symptoms by 4 years of age and
Jian-Ying Tian et al.
Experimental and therapeutic medicine, 12(2), 683-689 (2016-07-23)
The aim of the present study was to observe the effects of a general extract of Lycium bararum polysaccharides (LBPs) on methylmercury (MeHg)-induced damage in hippocampus neural stem cells (hNSCs). The hippocampal tissues of embryonic day 16 Sprague-Dawley rats were

Numéro d'article de commerce international

RéférenceGTIN
MAB562804053252727436

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