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L-Methionine

Name: <SC>L</SC>-Methionine
Brand: SAJ

SAJ special grade, ≥98.5%

Synonym(s):

(S)-2-Amino-4-(methylmercapto)butyric acid, L-2-Amino-4-(methylthio)butanoic acid

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About This Item

Linear Formula:

CH₃SCH₂CH₂CH(NH₂)CO₂H

CAS Number:

63-68-3

Molecular Weight:

149.21

EC Number:

200-562-9

UNSPSC Code:

12352112

PubChem Substance ID:

329751692

Beilstein/REAXYS Number:

1722294

MDL number:

MFCD00063097

Assay:

≥98.5%

Grade:

SAJ special grade

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Properties

grade

SAJ special grade

assay

≥98.5%

form

solid

availability

available only in Japan

dilution

(for analytical testing)

mp

284 °C (dec.) (lit.)

application(s)

peptide synthesis

SMILES string

CSCC[C@H](N)C(O)=O

InChI

1S/C5H11NO2S/c1-9-3-2-4(6)5(7)8/h4H,2-3,6H2,1H3,(H,7,8)/t4-/m0/s1

InChI key

FFEARJCKVFRZRR-BYPYZUCNSA-N

Description

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Storage Class

11 - Combustible Solids

wgk

WGK 1

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Gloves, type N95 (US)

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The in vivo sparing of methionine by cysteine in sulfur amino acid requirements in animal models and adult humans.

Ronald O Ball et al.

The Journal of nutrition, 136(6 Suppl), 1682S-1693S (2006-05-17)

Sulfur amino acid metabolism has been receiving increased attention because of the link to chronic diseases such as cardiovascular disease, Alzheimer's disease, and diabetes. In addition, the role of cysteine and optimal intakes for physiological substrates such as glutathione are

Fragmentation reactions of methionine-containing protonated octapeptides and fragment ions therefrom: an energy-resolved study.

Alex G Harrison

Journal of the American Society for Mass Spectrometry, 24(10), 1555-1564 (2013-08-15)

The fragmentation reactions of the MH(+) ions as well as the b7, a7, and a7* ions derived therefrom have been studied in detail for the octapeptides MAAAAAAA, AAMAAAAA, AAAAMAAA, and AAAAAAMA. Ionization was by electrospray using a QqToF mass spectrometer

Correction of the consequences of mitochondrial 3243A>G mutation in the MT-TL1 gene causing the MELAS syndrome by tRNA import into mitochondria.

Olga Z Karicheva et al.

Nucleic acids research, 39(18), 8173-8186 (2011-07-05)

Mutations in human mitochondrial DNA are often associated with incurable human neuromuscular diseases. Among these mutations, an important number have been identified in tRNA genes, including 29 in the gene MT-TL1 coding for the tRNA(Leu(UUR)). The m.3243A>G mutation was described

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