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Merck

06-953

Anti-phospho-FKHRL1 (Ser253) Antibody

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About This Item

UNSPSC Code:
12352203
NACRES:
NA.41
eCl@ss:
32160702
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antibody product type

primary antibodies

Quality Level

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Storage Class

10-13 - German Storage Class 10 to 13


Certificates of Analysis (COA)

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Deregulation of FOXO3A during prostate cancer progression.
Sanjeev Shukla,Meenakshi Shukla,Gregory T Maclennan,Pingfu Fu,Sanjay Gupta
International journal of oncology null
Ke-Jie Yin et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 26(8), 2290-2299 (2006-02-24)
Amyloid-beta peptide (Abeta)-induced death in cerebral endothelial cells (CECs) is preceded by mitochondrial dysfunction and signaling events characteristic of apoptosis. Mitochondria-dependent apoptosis engages Bcl-2 family proteins, especially the BH3-only homologues, which play a key role in initiating the apoptotic cascade.
JoAnne S Richards et al.
Molecular endocrinology (Baltimore, Md.), 16(3), 580-599 (2002-03-05)
Follicular development is dependent on both intraovarian growth regulatory factors, such as IGF-I and estrogen, as well as the pituitary gonadotropins, FSH and LH. Recently, we have shown that FSH impacts the IGF-I pathway via stimulation of the PI3K cascade
Walbert J Bakker et al.
The Journal of cell biology, 164(2), 175-184 (2004-01-22)
Erythropoiesis requires tight control of expansion, maturation, and survival of erythroid progenitors. Because activation of phosphatidylinositol-3-kinase (PI3K) is required for erythropoietin/stem cell factor-induced expansion of erythroid progenitors, we examined the role of the PI3K-controlled Forkhead box, class O (FoxO) subfamily
Paclitaxel-induced nuclear translocation of FOXO3a in breast cancer cells is mediated by c-Jun NH2-terminal kinase and Akt.
Sunters, A; Madureira, PA; Pomeranz, KM; Aubert, M; Brosens, JJ; Cook, SJ; Burgering et al.
Cancer Research null

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