F5879
Ammonium iron(III) citrate
reagent grade, powder
Synonym(s):
Ammonium ferric citrate, Ferric ammonium citrate
InChI
1S/C6H8O7.Fe.H3N/c7-3(8)1-6(13,5(11)12)2-4(9)10;;/h13H,1-2H2,(H,7,8)(H,9,10)(H,11,12);;1H3/q;+3;/p-3
InChI key
FRHBOQMZUOWXQL-UHFFFAOYSA-K
SMILES string
N.OC12CC(=O)O[Fe](OC(=O)C1)OC(=O)C2
grade
reagent grade
form
powder
composition
Iron, 16.5-18.5%
cation traces
Fe: 16.5-18.5%
Quality Level
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General description
Ammonium iron(III)citrate can be used as a reagent and iron precursor in several organic reactions and Fe3O4-based nanomaterial preparation. It can be prepared by treating Fe(OH)3 with citric acid and NH4OH in various ratios.
Application
Ammonium iron(III) citrate can be used as a reagent to prepare amphiphilic siderophore iron acinetoferrin complex from acinetoferrin in the presence of HEPES (4-(2-hydroxyethyl)-1-piperazineethanesulfonic acid) buffer. It can also be used as an iron precursor to prepare Fe3O4-carbon dots magnetic-fluorescent hybrid nanoparticles.
Storage Class
11 - Combustible Solids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
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Pengfei Wang et al.
Science advances, 5(4), eaau8038-eaau8038 (2019-04-17)
The in situ measurement of the distribution of biomolecules inside a cell is one of the important goals in life science. Among various imaging techniques, magnetic imaging (MI) based on the nitrogen-vacancy (NV) center in diamond provides a powerful tool
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Journal of neural transmission (Vienna, Austria : 1996), 120(1), 37-48 (2012-03-27)
Iron accumulation and iron-related oxidative stress are involved in several pathological conditions and provide a rationale for the development of iron chelators as novel promising therapeutic strategies. Thus, we have recently synthesized multifunctional non-toxic, brain permeable iron chelating compounds, M30
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Macrophages play a critical role at the crossroad between iron metabolism and immunity, being able to store and recycle iron derived from the phagocytosis of senescent erythrocytes. The way by which macrophages manage non-heme iron at physiological concentration is still
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Arteriosclerosis, thrombosis, and vascular biology, 31(3), 683-690 (2010-12-25)
Increased body iron stores and hepcidin have been hypothesized to promote atherosclerosis by inducing macrophage iron accumulation and release of cytokines, but direct demonstration in human cells is lacking. The aim of this study was to evaluate the effect of
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