239275
meta-Phosphoric acid
ACS reagent, chips, 33.5-36.5%
Synonym(s):
Metaphosphoric acid
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About This Item
Linear Formula:
(HPO3)n
CAS Number:
NACRES:
NA.21
PubChem Substance ID:
UNSPSC Code:
12352106
MDL number:
Concentration:
33.5-36.5%
Form:
chips
Grade:
ACS reagent
grade
ACS reagent
Quality Level
form
chips
contains
57.0-63.0% sodium phosphite as stabilizer
concentration
33.5-36.5%
pH
2 (33 g/L)
solubility
water: soluble
density
2 g/cm3
anion traces
MnO4- reducers: ≤0.02%, chloride (Cl-): ≤0.001%, nitrate (NO3-): ≤0.001%, sulfate (SO42-): ≤0.005%
cation traces
As: ≤1 ppm, Fe: ≤0.005%, heavy metals: ≤0.005% (by ICP)
SMILES string
OP(=O)=O
InChI
1S/HO3P/c1-4(2)3/h(H,1,2,3)
InChI key
UEZVMMHDMIWARA-UHFFFAOYSA-N
General description
meta-Phosphoric acid is a polymer form of phosphoric acid. It is used as a catalyst as well as a reactant in chemical synthesis.
Application
meta-Phosphoric acid can be used as:
- A catalyst in combination with InCl3 to synthesize 1,8-dioxooctahydroxanthene derivatives by the reaction of cyclic β-diketones and aldehydes.
- A phosphate precursor to synthesize antimony phosphate (SbPO4) by reacting with antimony metal at high temperatures.
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signalword
Danger
hcodes
Hazard Classifications
Eye Dam. 1 - Skin Corr. 1
Storage Class
8B - Non-combustible corrosive hazardous materials
flash_point_f
Not applicable
flash_point_c
Not applicable
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Chand Basha Davuljigari et al.
International journal of molecular sciences, 22(11) (2021-07-03)
Acrylamide is a well characterized neurotoxicant known to cause neuropathy and encephalopathy in humans and experimental animals. To investigate the role of nuclear factor erythroid 2-related factor 2 (Nrf2) in acrylamide-induced neuropathy, male C57Bl/6JJcl adult mice were exposed to acrylamide
Sellamuthu S Gounder et al.
PloS one, 7(9), e45697-e45697 (2012-10-03)
Aging promotes accumulation of reactive oxygen/nitrogen species (ROS/RNS) in cardiomyocytes, which leads to contractile dysfunction and cardiac abnormalities. These changes may contribute to increased cardiovascular disease in the elderly. Inducible antioxidant pathways are regulated by nuclear erythroid 2 p45-related factor
Alicia Madurga et al.
American journal of physiology. Lung cellular and molecular physiology, 306(7), L684-L697 (2014-02-11)
Arrested alveolarization is the pathological hallmark of bronchopulmonary dysplasia (BPD), a complication of premature birth. Here, the impact of systemic application of hydrogen sulfide (H2S) on postnatal alveolarization was assessed in a mouse BPD model. Exposure of newborn mice to