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Merck

S9663

Sodium arsenate dibasic heptahydrate

ACS reagent, ≥98%

Synonym(s):

Disodium hydrogen arsenate heptahydrate, di-Sodium hydrogen arsenate heptahydrate

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About This Item

Linear Formula:
Na2HAsO4 · 7H2O
CAS Number:
10048-95-0
Molecular Weight:
312.01
NACRES:
NB.24
PubChem Substance ID:
329824631
UNSPSC Code:
12352302
MDL number:
MFCD00149179
Assay:
≥98%, 98.0-102.0%
Grade:
ACS reagent
Form:
powder

Key Documents

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InChI

1S/AsH3O4.2Na.7H2O/c2-1(3,4)5;;;;;;;;;/h(H3,2,3,4,5);;;7*1H2/q;2*+1;;;;;;;/p-2

SMILES string

O.O.O.O.O.O.O.[Na+].[Na+].O[As]([O-])([O-])=O

InChI key

KOLXPEJIBITWIQ-UHFFFAOYSA-L

grade

ACS reagent

assay

≥98%, 98.0-102.0%

form

powder

impurities

≤0.005% Insoluble matter

pH

8.5-9 (25 °C, 50 g/L)

anion traces

arsenite (As2O3): ≤0.01%, chloride (Cl-): ≤0.001%, nitrate (NO3-): ≤0.005%, sulfate (SO42-): ≤0.01%

cation traces

Fe: ≤0.001%, heavy metals (as Pb): ≤0.002%

Quality Level

200

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Application

Sodium arsenate dibasic heptahydrate can be used as an internal standard for the quantification of arsenic species using various spectroscopic methods.

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Danger

Hazard Classifications

Acute Tox. 3 Inhalation - Acute Tox. 3 Oral - Aquatic Acute 1 - Aquatic Chronic 1 - Carc. 1A

Storage Class

6.1A - Combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

Regulatory Listings

Regulatory Listings are mainly provided for chemical products. Only limited information can be provided here for non-chemical products. No entry means none of the components are listed. It is the user’s obligation to ensure the safe and legal use of the product.

10048-95-0

CAS No.

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Certificates of Analysis (COA)

Lot/Batch Number
BCBM0939

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Shugo Suzuki et al.
Toxicology, 299(2-3), 155-159 (2012-06-06)
Inorganic arsenic is a known human carcinogen, inducing tumors of the skin, urinary bladder and lung. It is metabolized to organic methylated arsenicals. 2,3-Dimercaptopropane-1-sulfonic acid (DMPS), a chelating agent, is capable of reducing pentavalent arsenicals to the trivalent state and
M Sri Lakshmi Sunita et al.
Ecotoxicology (London, England), 21(1), 202-212 (2011-09-01)
In the present study, 44 arsenic-resistant bacteria were isolated through serial dilutions on agar plate with concentrations ≥0.05 mM of sodium arsenite and ≥10 mM of sodium arsenate from Mandovi and Zuari--estuarine water systems. The ars genotype characterization in 36
Deniz Yildiz et al.
Biological trace element research, 150(1-3), 451-459 (2012-08-15)
The objective of the present study was to investigate if arsenic exposure results in glutathione efflux from human erythrocytes. Arsenite significantly depleted intracellular nonprotein thiol level in a time- and concentration-dependent manner. The intracellular nonprotein thiol level was decreased to
Samantha L Goggin et al.
Neurotoxicology, 33(5), 1338-1345 (2012-09-11)
Over the past two decades, key advancements have been made in understanding the complex pathology that occurs following not only high levels of arsenic exposure (>1 ppm) but also levels previously considered to be low (<100 ppb). Past studies have
Takayuki Watanabe et al.
Archives of toxicology, 85(6), 577-588 (2011-05-04)
It has been suggested that arsenic (+3 oxidation state) methyltransferase (AS3MT) plays a critical role in methylation of arsenic, and that arsenic-glutathione conjugate is a substrate for AS3MT-catalyzed methylation of arsenic. However, the mechanism of arsenic methylation in cells is
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