B1385
Butoxamine hydrochloride
analytical standard, for drug analysis, mixture of diastereomers
Synonym(s):
α-(1-[t-Butylamino]ethyl)-2,5-dimethoxybenzyl alcohol
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About This Item
Empirical Formula (Hill Notation):
C15H25NO3 · HCl
CAS Number:
Molecular Weight:
303.82
NACRES:
NA.24
PubChem Substance ID:
UNSPSC Code:
41116107
EC Number:
227-169-5
MDL number:
Quality Level
technique(s)
HPLC: suitable, gas chromatography (GC): suitable
application(s)
forensics and toxicology
pharmaceutical (small molecule)
veterinary
format
neat
SMILES string
Cl.COc1ccc(OC)c(c1)C(O)C(C)NC(C)(C)C
InChI
1S/C15H25NO3.ClH/c1-10(16-15(2,3)4)14(17)12-9-11(18-5)7-8-13(12)19-6;/h7-10,14,16-17H,1-6H3;1H
InChI key
URPAECSKKQLCII-UHFFFAOYSA-N
General description
Butoxamine hydrochloride is a β-andrenoceptor antagonist, which shows β2-selectivity.
Application
Refer to the product′s Certificate of Analysis for more information on a suitable instrument technique. Contact Technical Service for further support.
signalword
Warning
hcodes
Hazard Classifications
Acute Tox. 4 Oral - Eye Irrit. 2
Storage Class
11 - Combustible Solids
wgk
WGK 3
ppe
dust mask type N95 (US), Eyeshields, Gloves
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Claudio Molinari et al.
Endocrinology, 148(8), 4080-4090 (2007-04-28)
Prolactin has been associated with many effects and has been implicated in the pathogenesis of pregnancy-related hypertensive disorders, although little is known about its vascular effects. The present study was designed to determine the primary effect of prolactin on regional
Nozomu Abe et al.
BioMed research international, 2020, 3214186-3214186 (2020-05-29)
Adrenaline quickly inhibits the release of histamine from mast cells. Besides β2-adrenergic receptors, several in vitro studies also indicate the involvement of α-adrenergic receptors in the process of exocytosis. Since exocytosis in mast cells can be detected electrophysiologically by the
Dong Zhang et al.
Pancreas, 38(1), 94-100 (2008-12-25)
Propranolol inhibited pancreatic cancer cell proliferation by blocking signaling through the beta-adrenoceptor. We hypothesized that propranolol may suppress pancreatic cancer cell growth through induction of apoptosis. The beta-adrenoceptor antagonist propranolol, beta1-adrenoceptor antagonist metoprolol, and beta2-adrenoceptor antagonist butoxamine were used to