04473
2,4-Pyridinedicarboxylic acid
≥98.0%
Synonym(s):
Lutidinic acid
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About This Item
Empirical Formula (Hill Notation):
C7H5NO4
CAS Number:
Molecular Weight:
167.12
NACRES:
NA.25
PubChem Substance ID:
UNSPSC Code:
12352106
EC Number:
207-892-2
MDL number:
Beilstein/REAXYS Number:
131631
Quality Level
assay
≥98.0%, 98.0-102.0% (T)
InChI
1S/C7H5NO4/c9-6(10)4-1-2-8-5(3-4)7(11)12/h1-3H,(H,9,10)(H,11,12)
InChI key
MJIVRKPEXXHNJT-UHFFFAOYSA-N
Application
2,4-Pyridinedicarboxylic acid is an in vitro and in cell inhibitor, as well as a known inhibitor of the histone lysine demethylases. 2,4-Pyridinedicarboxylic acid has been used in a study to determine that ruthenium(II) complexes exert antimetastatic effects on several tumor cell lines in vitro, achieved mostly by the effect on cell adhesion, migration and angiogenesis. . 2,4-Pyridinedicarboxylic acid has been used in a study to develop an assay that represents the first report of a RapidFire mass spectrometery assay for an epigenetics target.
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Storage Class
11 - Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
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Guo-liang Gu et al.
Spectrochimica acta. Part A, Molecular and biomolecular spectroscopy, 71(1), 209-214 (2008-02-19)
Two novel ligands containing two pyridine-2,6-dicarboxylic acid conjugative units, 4-(2-(2,6-dicarbox-ypyridin-4-yl)vinyl)pyridine-2,6-dicarboxylic acid (L1) and 4-(4-(2-(2,6-dicarboxypyridin-4-yl)vinyl)styryl)pyridine-2,6-dicarboxylic acid (L2) and their complexes with Tb(III) have been synthesized and characterized by elemental analysis, IR spectra and NMR. The ligand synthetic route was optimized and
H M Rowe et al.
Applied spectroscopy, 57(5), 532-537 (2003-12-09)
An adaptation of square-wave gated phase-modulation (GPM) fluorimetry allows for self-referenced intensity measurements without the complexity of dual excitation or dual emission wavelengths. This AC technique utilizes square-wave excitation, gated detection, a reference emitter, and a sensor molecule. The theory
Michael Raghunath et al.
Biochemical and biophysical research communications, 378(4), 766-771 (2008-12-11)
The rapid vascularisation of biomaterials and engineered tissue after implantation is a current unmet need. To this end, we explored the pharmacological option of inducing neovascularisation using compounds that inhibit hypoxia-induced factor-1alpha prolyl hydroxylase. This stabilises hypoxia inducible factor-1alpha and