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D9753

DL-3,4-Dihydroxyphenyl glycol

Name: <SC>DL</SC>-3,4-Dihydroxyphenyl glycol
Brand: SIGMA

Synonym(s):

DL-α,β,3,4-Tetrahydroxyphenethyl alcohol, DL-β,3,4-Trihydroxyphenethyl alcohol

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About This Item

Empirical Formula (Hill Notation):

C₈H₁₀O₄

CAS Number:

28822-73-3

Molecular Weight:

170.16

NACRES:

NA.25

PubChem Substance ID:

24894324

UNSPSC Code:

12352106

EC Number:

249-260-9

MDL number:

MFCD00004702

Form:

powder

Quality level:

200

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Properties

form

powder

Quality Level

200

storage temp.

−20°C

SMILES string

OCC(O)c1ccc(O)c(O)c1

InChI

1S/C8H10O4/c9-4-8(12)5-1-2-6(10)7(11)3-5/h1-3,8-12H,4H2

InChI key

MTVWFVDWRVYDOR-UHFFFAOYSA-N

Description

pictograms

GHS07

signalword

Warning

hcodes

H319

pcodes

P264 - P280 - P305 + P351 + P338 - P337 + P313

Hazard Classifications

Eye Irrit. 2

Storage Class

11 - Combustible Solids

wgk

WGK 3

ppe

dust mask type N95 (US), Eyeshields, Gloves

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Catecholamine metabolism: a contemporary view with implications for physiology and medicine.

Graeme Eisenhofer et al.

Pharmacological reviews, 56(3), 331-349 (2004-08-20)

This article provides an update about catecholamine metabolism, with emphasis on correcting common misconceptions relevant to catecholamine systems in health and disease. Importantly, most metabolism of catecholamines takes place within the same cells where the amines are synthesized. This mainly

Cerebrospinal fluid biomarkers of central catecholamine deficiency in Parkinson's disease and other synucleinopathies.

David S Goldstein et al.

Brain : a journal of neurology, 135(Pt 6), 1900-1913 (2012-03-28)

Central catecholamine deficiency characterizes α-synucleinopathies such as Parkinson's disease. We hypothesized that cerebrospinal fluid levels of neuronal metabolites of catecholamines provide neurochemical biomarkers of these disorders. To test this hypothesis we measured cerebrospinal fluid levels of catechols including dopamine, norepinephrine

Deficits in ventromedial prefrontal cortex group 1 metabotropic glutamate receptor function mediate resistance to extinction during protracted withdrawal from an extensive history of cocaine self-administration.

Osnat Ben-Shahar et al.

The Journal of neuroscience : the official journal of the Society for Neuroscience, 33(2), 495-506a-495-506a (2013-01-11)

Anomalies in prefrontal cortex (PFC) function are posited to underpin difficulties in learning to suppress drug-seeking behavior during abstinence. Because group 1 metabotropic glutamate receptors (mGluRs) regulate drug-related learning, we assayed the consequences of extended access to intravenous cocaine (6

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