HT901
Crystal Violet Solution
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About This Item
Empirical Formula (Hill Notation):
C25H30ClN3
CAS Number:
Molecular Weight:
407.98
UNSPSC Code:
12171500
NACRES:
NA.47
PubChem Substance ID:
MDL number:
Beilstein/REAXYS Number:
3580948
form
solution
Quality Level
shelf life
Expiry date on the label
IVD
for in vitro diagnostic use
concentration
2.3%
technique(s)
microbe id | staining: suitable
pH
4.6
application(s)
hematology
histology
storage temp.
room temp
SMILES string
[Cl-].CN(C)c1ccc(cc1)\C(c2ccc(cc2)N(C)C)=C3/C=C\C(C=C3)=[N+](/C)C
InChI
1S/C25H30N3.ClH/c1-26(2)22-13-7-19(8-14-22)25(20-9-15-23(16-10-20)27(3)4)21-11-17-24(18-12-21)28(5)6;/h7-18H,1-6H3;1H/q+1;/p-1
InChI key
ZXJXZNDDNMQXFV-UHFFFAOYSA-M
Application
Kit component of Gram Stain kits HT90A and HT90T (for staining of heat-fixed films and tissue, respectively).
Other Notes
HT901 is composed of 2.3% certified crystal violet, 0.1% ammonium oxalate monohydrate, and 20% ethyl alcohol, SD3A.
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signalword
Warning
hcodes
Hazard Classifications
Aquatic Chronic 3 - Carc. 2 - Eye Irrit. 2 - Flam. Liq. 3
Storage Class
3 - Flammable liquids
wgk
WGK 2
flash_point_f
100.0 °F - closed cup
flash_point_c
37.8 °C - closed cup
ppe
Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter
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W R Richter et al.
Journal of applied microbiology, 128(4), 1050-1059 (2019-11-30)
The purpose of this study was to evaluate the effects of altered environmental conditions on the persistence of Francisella tularensis bacteria and Venezuelan equine encephalitis virus (VEEV), on two material types. Francisella tularensis (F.t.) and VEEV were inoculated (c. 1 × 108 colony-forming
Chunmin Ge et al.
Hepatology (Baltimore, Md.), 69(6), 2608-2622 (2019-01-30)
Replication fork stability during DNA replication is vital for maintenance of genomic stability and suppression of cancer development in mammals. ATR (ataxia-telangiectasia mutated [ATM] and RAD3-related) is a master regulatory kinase that activates the replication stress response to overcome replication
Kai-Feng Hung et al.
International journal of biological sciences, 15(5), 1080-1090 (2019-06-12)
Up-regulation of ASB6 has been previously associated with late-stage and poor prognosis of oral squamous cell carcinoma (OSCC) patients. To explore the cellular and molecular basis of how ASB6 enhances the malignancy of OSCC, we employed the clonogenicity and migration