M0518
Minimum Essential Medium Eagle
Joklik Modification, with L-glutamine, without calcium chloride and sodium bicarbonate, suitable for cell culture
Synonym(s):
JMEM, Joklik Modified MEM, MEM
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About This Item
NACRES:
NA.75
UNSPSC Code:
12352207
Product Name
Minimum Essential Medium Eagle, Joklik Modification, with L-glutamine, without calcium chloride and sodium bicarbonate, suitable for cell culture
form
powder
technique(s)
cell culture | mammalian: suitable
components
HEPES: no
sodium pyruvate: no
L-glutamine: yes
NaHCO3: no
phenol red: yes
shipped in
ambient
storage temp.
2-8°C
Quality Level
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Application
Recommended for suspension cultures.
Preparation Note
Formulated to contain 11.0 grams of powder per liter of medium.
Supplement with 2.0 g/L sodium bicarbonate.
Storage Class
11 - Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
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Molecular bioSystems, 8(9), 2351-2359 (2012-06-28)
Although angiotensin (Ang)II-induced Janus-activated kinase (JAK)2 phosphorylation was reported to be enhanced in failing human cardiomyocytes, the downstream balance between cardio-protective (signal transducer and activator of transcription-STAT3) and the pro-inflammatory (STAT2 and STAT5) response remains unexplored. Therefore STATs phosphorylation and
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Despite their important role in metastatic disease, no general method to detect circulating stromal cells (CStCs) exists. Here, we present the Metabolic Assay-Chip (MA-Chip) as a label-free, droplet-based microfluidic approach allowing single-cell extracellular pH measurement for the detection and isolation
Winston T Stauffer et al.
International journal of molecular sciences, 21(4) (2020-02-23)
Activating transcription factor-6 α (ATF6) is one of the three main sensors and effectors of the endoplasmic reticulum (ER) stress response and, as such, it is critical for protecting the heart and other tissues from a variety of environmental insults
Christopher A Drummond et al.
Physiological genomics, 48(3), 220-229 (2015-12-25)
Chronic kidney disease (CKD) is accompanied by cardiac fibrosis, hypertrophy, and dysfunction, which are commonly referred to as uremic cardiomyopathy. Our previous studies found that Na/K-ATPase ligands or 5/6th partial nephrectomy (PNx) induces cardiac fibrosis in rats and mice. The
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