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NACRES:
NA.32
UNSPSC Code:
41116158
Gene information:
human ... AKT1(207)
Input:
sample type: human cell lysate
sample type tissue lysate
sample type tissue lysate
Species reactivity:
human, mouse, rat
Storage temp.:
−20°C
Shipped in:
wet ice
Servicio técnico
¿Necesita ayuda? Nuestro equipo de científicos experimentados está aquí para ayudarle.
Permítanos ayudarlespecies reactivity
human, mouse, rat
packaging
kit of 96 wells (12 strips x 8 wells)
technique(s)
ELISA: suitable, capture ELISA: suitable
input
sample type: human cell lysate
sample type tissue lysate
detection method
colorimetric
shipped in
wet ice
storage temp.
−20°C
Gene Information
human ... AKT1(207)
General description
The Phospho-Akt (pSer473) ELISA (Enzyme-Linked Immunosorbent Assay) kit is a very rapid, convenient and sensitive assay kit that can monitor the activation or function of important biological pathways in cell lysates.
Immunogen
Akt (pSer473) synthetic phosphopeptide, Recombinant Human EGFR
Application
For research use only. Not for use in diagnostic procedures.
Please refer to the attached General ELISA KIT Procedure (sandwich, competitive & Indirect ELISA)
Please refer to the attached General ELISA KIT Procedure (sandwich, competitive & Indirect ELISA)
Los componentes del kit también están disponibles por separado
Referencia del producto
Descripción
SDS & Precios
signalword
Warning
hcodes
pcodes
Hazard Classifications
Met. Corr. 1
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
Clase de almacenamiento
12 - Non Combustible Liquids
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International journal of molecular sciences, 19(8) (2018-08-22)
The effects of Ca2+-activated K⁺ (BK) channel modulation by Paxilline (PAX) (10-7⁻10-4 M), Iberiotoxin (IbTX) (0.1⁻1 × 10-6 M) and Resveratrol (RESV) (1⁻2 × 10-4 M) on cell cycle and proliferation, AKT1pSer473 phosphorylation, cell diameter, and BK currents were investigated
Molly Stanley et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 36(46), 11704-11715 (2016-11-18)
Hyperinsulinemia is a risk factor for late-onset Alzheimer's disease (AD). In vitro experiments describe potential connections between insulin, insulin signaling, and amyloid-β (Aβ), but in vivo experiments are needed to validate these relationships under physiological conditions. First, we performed hyperinsulinemic-euglycemic
