610046
Boron-11B
95 atom % 11B
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A propos de cet article
Formule linéaire :
11B
Numéro CAS:
Poids moléculaire :
11.01
UNSPSC Code:
12141907
PubChem Substance ID:
MDL number:
Isotopic purity:
95 atom % 11B
Mass shift:
depleted
Form:
solid
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1S/B/i1+0
SMILES string
[11B]
InChI key
ZOXJGFHDIHLPTG-IGMARMGPSA-N
isotopic purity
95 atom % 11B
form
solid
mass shift
depleted
Catégories apparentées
Packaging
This product may be available from bulk stock and can be packaged on demand. For information on pricing, availability and packaging, please contact Stable Isotopes Customer Service.
signalword
Warning
hcodes
Hazard Classifications
Acute Tox. 4 Oral - Aquatic Chronic 3
Classe de stockage
11 - Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
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John C Rose et al.
Nature methods, 14(9), 891-896 (2017-07-25)
We developed a chemically inducible Cas9 (ciCas9) and a droplet digital PCR assay for double-strand breaks (DSB-ddPCR) to investigate the kinetics of Cas9-mediated generation and repair of DSBs in cells. ciCas9 is a rapidly activated, single-component Cas9 variant engineered by
Chathura D Suraweera et al.
Cell death & disease, 9(10), 967-967 (2018-09-22)
Bcl-2 family proteins play a crucial role in regulating apoptosis, a process critical for development, eliminating damaged or infected cells, host-pathogen interactions and in disease. Dysregulation of Bcl-2 proteins elicits an expansive cell survival mechanism promoting cell migration, invasion and
S Matt Gilbert et al.
Scientific reports, 7(1), 15096-15096 (2017-11-10)
We demonstrate the fabrication of individual nanopores in hexagonal boron nitride (h-BN) with atomically precise control of the pore shape and size. Previous methods of pore production in other 2D materials typically create pores with irregular geometry and imprecise diameters.
Michael Fricker et al.
Physiological reviews, 98(2), 813-880 (2018-03-01)
Neuronal cell death occurs extensively during development and pathology, where it is especially important because of the limited capacity of adult neurons to proliferate or be replaced. The concept of cell death used to be simple as there were just
Yulia Kushnareva et al.
PLoS biology, 10(9), e1001394-e1001394 (2012-10-11)
Bax/Bak-mediated mitochondrial outer membrane permeabilization (MOMP) is essential for "intrinsic" apoptotic cell death. Published studies used synthetic liposomes to reveal an intrinsic pore-forming activity of Bax, but it is unclear how other mitochondrial outer membrane (MOM) proteins might facilitate this
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