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Merck

SAB5600066

Anti-AKT1 antibody, Rabbit monoclonal

recombinant, expressed in HEK 293 cells, clone RM252, purified immunoglobulin

Synonym(s):

Serine/Threonine Kinase 1

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About This Item

NACRES:
NA.41
UNSPSC Code:
12352203

Product Name

Anti-AKT1 antibody, Rabbit monoclonal, recombinant, expressed in HEK 293 cells, clone RM252, purified immunoglobulin

recombinant

expressed in HEK 293 cells

conjugate

unconjugated

antibody form

purified immunoglobulin

antibody product type

primary antibodies

clone

RM252, monoclonal
recombinant monoclonal

form

buffered aqueous glycerol solution

species reactivity

human

technique(s)

immunoblotting: 1:1000-1:2000
immunohistochemistry: 1:500-1:1000

isotype

IgG

NCBI accession no.

UniProt accession no.

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Quality Level

Gene Information

human ... AKT1(207)

Biochem/physiol Actions

This antibody reacts to human Akt1. This antibody may also react to bovine, mouse or rat Akt1, as predicted by immunogen homology

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

Features and Benefits

Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.

Immunogen

Synthetic peptide corresponding to the C-terminus of human Akt1

Physical form

Solution in phosphate buffered saline containing 50% glycerol, 1% BSA and 0.09% sodium azide.

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Storage Class

10 - Combustible liquids

wgk

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable


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Aniketh Bishnu et al.
Cell death & disease, 12(2), 161-161 (2021-02-10)
Alterations in key kinases and signaling pathways can fine-tune autophagic flux to promote the development of chemoresistance. Despite empirical evidences of strong association between enhanced autophagic flux with acquired chemoresistance, it is still not understood whether an ongoing autophagic flux
Arijit Mal et al.
Frontiers in cell and developmental biology, 8, 597673-597673 (2021-01-26)
Substantial number of breast cancer (BC) patients undergoing radiation therapy (RT) develop local recurrence over time. During RT therapy, cells can gradually acquire resistance implying adaptive radioresistance. Here we probe the mechanisms underlying this acquired resistance by first establishing radioresistant

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