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Merck

SAB5600242

Anti-CK-5/CK-6 (Cytokeratin 5/ Cytokeratin 6) antibody, Rabbit monoclonal

recombinant, expressed in HEK 293 cells, clone RM341, purified immunoglobulin

Synonym(s):

Keratin 5, Keratin 6

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About This Item

NACRES:
NA.41
UNSPSC Code:
12352203
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Product Name

Anti-CK-5/CK-6 (Cytokeratin 5/ Cytokeratin 6) antibody, Rabbit monoclonal, recombinant, expressed in HEK 293 cells, clone RM341, purified immunoglobulin

recombinant

expressed in HEK 293 cells

conjugate

unconjugated

antibody form

purified immunoglobulin

antibody product type

primary antibodies

clone

RM341, monoclonal
recombinant monoclonal

form

buffered aqueous glycerol solution

species reactivity

human

technique(s)

immunohistochemistry: 1:500—1:1000
western blot: 1:2000-1:4000

isotype

IgG

NCBI accession no.

UniProt accession no.

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Gene Information

human ... KRT5(3852)

Biochem/physiol Actions

This antibody reacts to both human CK-5 (Cytokeratin 5) and CK-6 (Cytokeratin 6).

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

Immunogen

A peptide corresponding to the C-terminus of Human Cytokeratin 5

Physical form

Solution in phosphate buffered saline containing 50% Glycerol/PBS with 1% BSA and 0.09% sodium azide.

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Storage Class

10 - Combustible liquids

wgk

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable


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Jun Fan et al.
Frontiers in oncology, 11, 740410-740410 (2021-12-21)
Craniopharyngiomas (CPs) predominantly involving the third ventricle were commonly termed "intraventricular" lesions. The aim of this study was to clarify the anatomical relationship between the tumor and the third ventricle by both surgical and histological investigation. A retrospective review of
NOTCH4I?L12_16 sensitizes lung adenocarcinomas to EGFR-TKIs through transcriptional down-regulation of HES1.
Zhang, et al.
Nature Communications, 14, 3183-3183 (2023)

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