299367
Urea-12C
99.9 atom % 12C
別名:
Carbamide-12C
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この商品について
化学式:
H2N12CONH2
CAS番号:
分子量:
60.04
UNSPSC Code:
12352111
PubChem Substance ID:
EC Number:
200-315-5
MDL number:
製品名
Urea-12C, 99.9 atom % 12C
InChI key
XSQUKJJJFZCRTK-IGMARMGPSA-N
InChI
1S/CH4N2O/c2-1(3)4/h(H4,2,3,4)/i1+0
SMILES string
N[12C](N)=O
description
13C-depleted
isotopic purity
99.9 atom % 12C
form
solid
mp
132-135 °C (lit.)
mass shift
depleted
Quality Level
Gene Information
human ... CA1(759), CA2(760), EPHX2(2053)
mouse ... Ephx2(13850)
rat ... Ppm1a(24666)
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Packaging
This product may be available from bulk stock and can be packaged on demand. For information on pricing, availability and packaging, please contact Stable Isotopes Customer Service.
保管分類
11 - Combustible Solids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type N95 (US)
Giora van Straten et al.
PloS one, 9(6), e100077-e100077 (2014-06-20)
The detoxification of ammonia occurs mainly through conversion of ammonia to urea in the liver via the urea cycle and glutamine synthesis. Congenital portosystemic shunts (CPSS) in dogs cause hyperammonemia eventually leading to hepatic encephalopathy. In this study, the gene
Julian Wampfler et al.
Journal of leukocyte biology, 98(3), 357-363 (2015-05-21)
Successful myeloid differentiation depends on the expression of a series of miRNAs. Thus, it is hardly surprising that miRNAs are globally repressed in AML, a disease mainly characterized by a block in cellular myeloid differentiation. Studies investigating the mechanisms for
Anne-Roos S Frenay et al.
PloS one, 10(6), e0129732-e0129732 (2015-06-11)
Some diseases associated with a temporary deterioration in kidney function and/or development of proteinuria show an apparently complete functional remission once the initiating trigger is removed. While it was earlier thought that a transient impairment of kidney function is harmless
Hannah M Kaneb et al.
Human molecular genetics, 24(5), 1363-1373 (2014-10-26)
Amyotrophic lateral sclerosis (ALS) is a fatal neurodegenerative disorder characterized by the selective death of motor neurons. Causative mutations in the global RNA-processing proteins TDP-43 and FUS among others, as well as their aggregation in ALS patients, have identified defects
Jean-Ha Baek et al.
Human molecular genetics, 24(5), 1305-1321 (2014-10-26)
Hutchinson-Gilford progeria syndrome (HGPS) is a segmental progeroid syndrome with multiple features suggestive of premature accelerated aging. Accumulation of progerin is thought to underlie the pathophysiology of HGPS. However, despite ubiquitous expression of lamin A in all differentiated cells, the
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