28-3030
水酸化ナトリウム 溶液
0.2 M
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この商品について
化学式:
NaOH
CAS番号:
分子量:
40.00
UNSPSC Code:
12352106
PubChem Substance ID:
MDL number:
Concentration:
0.2 M, 1/5 N
Form:
liquid
InChI
1S/Na.H2O/h;1H2/q+1;/p-1
SMILES string
[OH-].[Na+]
InChI key
HEMHJVSKTPXQMS-UHFFFAOYSA-M
vapor pressure
3 mmHg ( 37 °C)
form
liquid
availability
available only in Japan
concentration
0.2 M, 1/5 N
density
1 g/cm3 at 20 °C
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関連するカテゴリー
signalword
Warning
hcodes
Hazard Classifications
Eye Irrit. 2 - Met. Corr. 1 - Skin Irrit. 2
保管分類
8B - Non-combustible corrosive hazardous materials
wgk
nwg
flash_point_f
Not applicable
flash_point_c
Not applicable
適用法令
試験研究用途を考慮した関連法令を主に挙げております。化学物質以外については、一部の情報のみ提供しています。 製品を安全かつ合法的に使用することは、使用者の義務です。最新情報により修正される場合があります。WEBの反映には時間を要することがあるため、適宜SDSをご参照ください。
28-3030-5-500ML-J:
jan
Triggered Ca2+ influx is required for extended synaptotagmin 1-induced ER-plasma membrane tethering.
Olof Idevall-Hagren et al.
The EMBO journal, 34(17), 2291-2305 (2015-07-24)
The extended synaptotagmins (E-Syts) are ER proteins that act as Ca(2+)-regulated tethers between the ER and the plasma membrane (PM) and have a putative role in lipid transport between the two membranes. Ca(2+) regulation of their tethering function, as well
Horacio Cardenas et al.
Epigenetics, 9(11), 1461-1472 (2014-12-04)
A key step in the process of metastasis is the epithelial-to-mesenchymal transition (EMT). We hypothesized that epigenetic mechanisms play a key role in EMT and to test this hypothesis we analyzed global and gene-specific changes in DNA methylation during TGF-β-induced
Aparajita H Chourasia et al.
EMBO reports, 16(9), 1145-1163 (2015-08-02)
BNip3 is a hypoxia-inducible protein that targets mitochondria for autophagosomal degradation. We report a novel tumor suppressor role for BNip3 in a clinically relevant mouse model of mammary tumorigenesis. BNip3 delays primary mammary tumor growth and progression by preventing the
Joshua S McLane et al.
Biophysical journal, 109(2), 249-264 (2015-07-23)
Mechanical properties of the tumor microenvironment have emerged as key factors in tumor progression. It has been proposed that increased tissue stiffness can transform stromal fibroblasts into carcinoma-associated fibroblasts. However, it is unclear whether the three to five times increase
Johannes Egerer et al.
The Journal of investigative dermatology, 135(10), 2368-2376 (2015-05-23)
Gerodermia osteodysplastica is a hereditary segmental progeroid disorder affecting skin, connective tissues, and bone that is caused by loss-of-function mutations in GORAB. The golgin, RAB6-interacting (GORAB) protein localizes to the Golgi apparatus and interacts with the small GTPase RAB6. In
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