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Fórmula empírica (notación de Hill):
C6H12O6
Número CAS:
Peso molecular:
180.16
UNSPSC Code:
12352201
NACRES:
NA.25
PubChem Substance ID:
EC Number:
239-630-8
Beilstein/REAXYS Number:
1724622
MDL number:
Servicio técnico
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Permítanos ayudarleQuality Level
assay
≥99% (HPLC)
form
powder
technique(s)
HPLC: suitable
color
white
solubility
water: 50 mg/mL, clear, colorless
SMILES string
OC[C@@H]1OC(O)[C@@H](O)[C@H](O)[C@@H]1O
InChI
1S/C6H12O6/c7-1-2-3(8)4(9)5(10)6(11)12-2/h2-11H,1H2/t2-,3+,4+,5-,6?/m0/s1
InChI key
WQZGKKKJIJFFOK-DHVFOXMCSA-N
Biochem/physiol Actions
L-Galactose was shown to be a key intermediate in the molecular pathway of converting D-glucose to oxalic acid in Pistia stratiotes.
Other Notes
To gain a comprehensive understanding of our extensive range of Monosaccharides for your research, we encourage you to visit our Carbohydrates Category page.
Clase de almacenamiento
11 - Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type N95 (US)
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Minoru Tomizawa et al.
Oncology letters, 14(1), 899-902 (2017-07-12)
Tissues surrounding hepatocellular carcinomas (HCCs) lack glucose. Hepatocyte selection medium (HSM) is deficient in glucose and is supplemented with galactose. HCC cells were cultured in HSM to investigate the stem cell markers α-fetoprotein (AFP) and cluster of differentiation 44 (CD44).
S E Keates et al.
Phytochemistry, 53(4), 433-440 (2000-03-24)
Axenic Pistia stratiotes L. plants were pulse-chase labeled with [14C]oxalic acid, L[1-14C]ascorbic acid, L-6-14C]ascorbic acid, D-[1-14C]erythorbic acid, L-[1-14C]galactose, or [1-14C]glycolate. Specific radioactivities of L-ascorbic acid (AsA), free oxalic acid (OxA) and calcium oxalate (CaOx) in labeled plants were compared. Samples
Jolene M Garber et al.
Communications biology, 3(1), 2-2 (2020-01-12)
Although the gastrointestinal pathogen Campylobacter jejuni was considered asaccharolytic, >50% of sequenced isolates possess an operon for L-fucose utilization. In C. jejuni NCTC11168, this pathway confers L-fucose chemotaxis and competitive colonization advantages in the piglet diarrhea model, but the catabolic