SAB5200009
Monoclonal Anti-Nitrotyrosine antibody produced in mouse
clone 39B6, 1 mg/mL, purified immunoglobulin
Sinónimos:
Anti-N/A, Anti-Nitrotyrosine
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NACRES:
NA.41
UNSPSC Code:
12352203
Conjugate:
unconjugated
Clone:
39B6, monoclonal
Application:
ELISA (i), IHC, IP, WB
Citations:
5
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Permítanos ayudarlebiological source
mouse
conjugate
unconjugated
antibody form
purified immunoglobulin
antibody product type
primary antibodies
clone
39B6, monoclonal
form
buffered aqueous glycerol solution
species reactivity
mouse, dog, rat, human
concentration
1 mg/mL
technique(s)
immunohistochemistry: suitable, immunoprecipitation (IP): suitable, indirect ELISA: suitable, western blot: suitable
isotype
IgG2a
shipped in
wet ice
storage temp.
−20°C
target post-translational modification
nitration (Tyr)
Quality Level
Gene Information
human ... NOS1(4842)
Immunogen
Hybridoma line 39B6
Biochem/physiol Actions
Recognizes 3-nitrotyrosine moieties. No detectable cross-reactivity with non-nitrated tyrosine. Not species specific.
Features and Benefits
Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.
Physical form
Solution in PBS, 50% glycerol, and 0.09% sodium azide
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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Clase de almacenamiento
10 - Combustible liquids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
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Pieter C Vogelaar et al.
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Antonella Tramutola et al.
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Down syndrome (DS) is the most frequent genetic cause of intellectual disability and is strongly associated with Alzheimer's disease (AD). Brain insulin resistance greatly contributes to AD development in the general population and previous studies from our group showed an
Qingyi Ma et al.
Molecular neurobiology, 56(8), 5608-5625 (2019-01-19)
Neonatal hypoxic-ischemic (HI) brain injury causes significant mortality and long-term neurologic sequelae. We previously demonstrated that HI significantly increased microRNA-210 (miR-210) in the neonatal rat brain and inhibition of brain endogenous miR-210 was neuroprotective in HI brain injury. However, the
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