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524632

Phosphatase Inhibitor Cocktail V

50X, lyophilized solid, for the inhibition of acid, alkaline, serine/threonine and protein tyrosine phosphatases. This small molecule/inhibitor is primarily used for Phosphorylation & Dephosphorylation applications.

Synonyme(s) :

Phosphatase inhibitor cocktail

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A propos de cet article

UNSPSC Code:
12352200
NACRES:
NA.54
Form:
lyophilized solid
Solubility:
water: soluble
Storage temp.:
2-8°C
Service technique
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Nom du produit

Phosphatase Inhibitor Cocktail Set V, 50X, Lyophilized, The Phosphatase Inhibitor Cocktail Set V, 50X, Lyophilized controls the activity of Phosphatase. This small molecule/inhibitor is primarily used for Phosphorylation & Dephosphorylation applications.

form

lyophilized solid

manufacturer/tradename

Calbiochem®

storage condition

OK to freeze, desiccated (hygroscopic), protect from light

solubility

water: soluble

shipped in

ambient

storage temp.

2-8°C

General description

A cocktail of four phosphatase inhibitors for broad-spectrum inhibition of acid, alkaline, serine/threonine and protein tyrosine phosphatases. Available as a 1 ml vial or a set of five 1 ml vials. Each vial, when reconstituted with 1 ml H2O contains 250 mM Sodium Fluoride, 50 mM β-Glycerophosphate (Cat. No. 35675), 50 mM Sodium Pyrophosphate Decahydrate, 50 mM Sodium Orthovanadate.

Packaging

Packaged under inert gas

Preparation Note

Following reconstitution, aliquot and freeze (-20°C) for long-term storage. Dilute 1:50 just prior to use.
Reconstitute each vial with 1 ml H₂O.

Legal Information

CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany

Disclaimer

Toxicity: Toxic (F)

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GHS05,GHS07

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Danger

Hazard Classifications

Acute Tox. 4 Oral - Eye Dam. 1 - Skin Irrit. 2

supp_hazards

EUH032

Classe de stockage

11 - Combustible Solids

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

Certificats d'analyse (COA)

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Bernd Stratmann et al.
International journal of molecular sciences, 23(13) (2022-07-10)
An oversupply of nutrients with a loss of metabolic flexibility and subsequent cardiac dysfunction are hallmarks of diabetic cardiomyopathy. Even if excess substrate is offered, the heart suffers energy depletion as metabolic fluxes are diminished. To study the effects of