449172
塩化マグネシウム
AnhydroBeads™, −10 mesh, 99.99% trace metals basis
別名:
マグノゲン
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この商品について
化学式:
MgCl2
CAS番号:
分子量:
95.21
NACRES:
NA.23
PubChem Substance ID:
UNSPSC Code:
12352302
EC Number:
232-094-6
MDL number:
Assay:
99.99% trace metals basis
Grade:
synthesis grade
製品名
塩化マグネシウム, AnhydroBeads™, −10 mesh, 99.99% trace metals basis
InChI key
TWRXJAOTZQYOKJ-UHFFFAOYSA-L
InChI
1S/2ClH.Mg/h2*1H;/q;;+2/p-2
SMILES string
Cl[Mg]Cl
grade
synthesis grade
product line
AnhydroBeads™
assay
99.99% trace metals basis
reaction suitability
core: magnesium
impurities
≤150.0 ppm Trace Metal Analysis
particle size
−10 mesh
mp
714 °C (lit.)
density
2.32 g/mL at 25 °C (lit.)
application(s)
electroplating
material synthesis precursor
Quality Level
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General description
塩化マグネシウムは、多硫化Mgフロー電池用の電解質溶液を調製する出発物質として使用できます。
Legal Information
AnhydroBeads is a trademark of Sigma-Aldrich Co. LLC
保管分類
13 - Non Combustible Solids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type N95 (US)
Role of chloride for a simple, non-grignard Mg electrolyte in ether-based solvents
Niya Sa, et al
ACS Applied Materials & Interfaces, 8, 16002-16008 (2016)
Stability and Disproportionation of Magnesium Polysulfides and the Effects on the Mg-Polysulfide Flow Battery
Peng He, et al.
Journal of the Electrochemical Society, 168, 110516-110516 (2021)
Manal A Farg et al.
Human molecular genetics, 23(13), 3579-3595 (2014-02-20)
Intronic expansion of a hexanucleotide GGGGCC repeat in the chromosome 9 open reading frame 72 (C9ORF72) gene is the major cause of familial amyotrophic lateral sclerosis (ALS) and frontotemporal dementia. However, the cellular function of the C9ORF72 protein remains unknown.
Fernando Cartón-García et al.
Scientific reports, 5, 12312-12312 (2015-07-24)
Inherited MYO5B mutations have recently been associated with microvillus inclusion disease (MVID), an autosomal recessive syndrome characterized by intractable, life-threatening, watery diarrhea appearing shortly after birth. Characterization of the molecular mechanisms underlying this disease and development of novel therapeutic approaches
Benjamin Dombert et al.
PloS one, 9(10), e110846-e110846 (2014-10-23)
Spinal muscular atrophy (SMA) is caused by deficiency of the ubiquitously expressed survival motoneuron (SMN) protein. SMN is crucial component of a complex for the assembly of spliceosomal small nuclear ribonucleoprotein (snRNP) particles. Other cellular functions of SMN are less
プロトコル
Accurately measure the moisture content in Magnesium Chloride (MgCl2) through Karl Fischer titration, using both Volumetric and Coulometric methods.
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