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Merck

RAB0229

Mouse IGF-I ELISA Kit

for serum, plasma and cell culture supernatant

Synonyme(s) :

Insulin-like growth factor I, Mechano growth factor, Somatomedin-C

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About This Item

NACRES:
NA.32
UNSPSC Code:
41116158

Nom du produit

Mouse IGF-I ELISA Kit, for serum, plasma and cell culture supernatant

species reactivity

mouse

packaging

kit of 96 wells (12 strips x 8 wells)

technique(s)

ELISA: suitable
capture ELISA: suitable

input

sample type cell culture supernatant(s)
sample type serum
sample type plasma

assay range

inter-assay cv: <12%
intra-assay cv: <10%
sensitivity: 4 pg/mL
standard curve range: 2.74-2000 pg/mL

detection method

colorimetric

shipped in

wet ice

storage temp.

−20°C

Gene Information

mouse ... Igf1(16000)

Catégories apparentées

Application

For research use only. Not for use in diagnostic procedures.
Mouse IGF-I ELISA kit has been used to measure plasma IGF-I (Insulin-like growth factor-I) level from Igf1r L/L and BAT-specific Igf1r-knockout (BATIGFIRKO) mice.
For research use only. Not for use in diagnostic procedures.
Please refer to the attached General ELISA KIT Procedure (sandwich, competitive & Indirect ELISA)
Mouse IGF-I ELISA Kit has been used to measure the concentration of insulin-like growth factor(IGF) secreted by mesenchymal stem cells(MSCs). It has also been used to measure the level of IGF1 in blood samples of mice.

Biochem/physiol Actions

IGF-I (Insulin-like growth factor-I) is required for fetal growth and development. It is also involved in the growth and repair of heart tissues. IGF-I has been linked to neuroplasticity and hippocampal neurogenesis. It is important in the recovery from ischaemic injury. IGF-I also facilitates the differentiation of adipocytes and other cells.

General description

The Mouse IGF-I ELISA (Enzyme-Linked Immunosorbent Assay) kit is an in vitro enzyme-linked immunosorbent assay for the quantitative measurement of mouse IGF-I in serum, plasma, cell culture supernatants and urine.

Immunogen

Recombinant Mouse IGF-I

Other Notes

A sample Certificate of Analysis is available for this product.
Please type the word sample in the text box provided for lot number.

pictograms

Corrosion
GHS05

signalword

Warning

hcodes

H290

Hazard Classifications

Met. Corr. 1

Classe de stockage

8A - Combustible corrosive hazardous materials

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Certificats d'analyse (COA)

Lot/Batch Number
0228K0484
1116J0484
1110J0484
0627J0484
0622J0484

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Consulter la Bibliothèque de documents

Flavia R Siqueira et al.
Physiology & behavior, 154, 68-75 (2015-11-26)
A low-salt (LS) diet during pregnancy has been linked to insulin resistance in adult offspring, at least in the experimental setting. However, it remains unclear if this effect is due to salt restriction during early or late pregnancy. To better
He He et al.
Inflammation, 38(6), 2178-2184 (2015-06-25)
Sepsis-induced myocardial injury (SIMI) is caused by various mechanisms. The aim of this study was to investigate the effects of salidroside (Sal) on SIMI and its mechanisms in rats. The sepsis model was established by intraperitoneal injection of lipopolysaccharide (LPS)
María Llorens-Martín et al.
The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry, 15(2), 134-148 (2009-03-25)
This review addresses the role of serum insulin-like growth factor 1 (IGF1) as one mechanism of adult neural plasticity, specifically, its regulation of hippocampal neurogenesis among other plasticity-related processes. It is suggested that IGF has been reused advantageously both for
Sebastian Mathes et al.
Journal of molecular medicine (Berlin, Germany), 100(11), 1647-1658 (2022-10-01)
Skeletal muscle exerts many beneficial effects on the human body including the contraction-dependent secretion of peptides termed myokines. We have recently connected the myokine secreted protein acidic and rich in cysteine (SPARC) to the formation of intramuscular adipose tissue (IMAT)
Márcio Buffolo et al.
Cell reports, 29(2), 270-282 (2019-10-10)
Accumulation of visceral (VIS) is a predictor of metabolic disorders and insulin resistance. This is due in part to the limited capacity of VIS fat to buffer lipids allowing them to deposit in insulin-sensitive tissues. Mechanisms underlying selective hypertrophic growth
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